Literatur

Ausgewählte Publikationen der Arbeitsgruppe (sortiert nach Erstautor)

  • Besgen P, Trommler P, Vollmer S, Prinz JC (2010) Ezrin, maspin, peroxiredoxin 2, and heat shock protein 27: potential targets of a streptococcal-induced autoimmune response in psoriasis. J Immunol 184: 5392-402
  • Diluvio L, Vollmer S, Besgen P, Ellwart JW, Chimenti S, Prinz JC (2006) Identical TCR beta-chain rearrangements in streptococcal angina and skin lesions of patients with psoriasis vulgaris. J Immunol 176: 7104-11
  • Ghoreschi K, Thomas P, Breit S, Dugas M, Mailhammer R, van Eden W, van der Zee R, Biedermann T, Prinz J, Mack M, Mrowietz U, Christophers E, Schlondorff D, Plewig G, Sander CA, Rocken M (2003) Interleukin-4 therapy of psoriasis induces Th2 responses and improves human autoimmune disease. Nat Med 9: 40-6
  • Kim SM, Bhonsle L, Besgen P, Nickel J, Backes A, Held K, Vollmer S, Dornmair K, Prinz JC (2012) Analysis of the Paired TCR alpha- and beta-chains of Single Human T Cells. PLoS ONE 7: e37338
  • Menssen A, Trommler P, Vollmer S, Schendel D, Albert E, Gurtler L, Riethmuller G, Prinz JC (1995) Evidence for an antigen-specific cellular immune response in skin lesions of patients with psoriasis vulgaris. J Immunol 155: 4078-83
  • Menssen A, Vollmer S, Trommler P, Sander C, Prinz JC (2000) Analysis of the TCRBV repertoire of T cells in normal, human skin: evidence for a restricted diversity. J Invest Dermatol 115: 66-73
  • Molin S, Ruzicka T, Prinz JC (2010) Bell's palsy during etanercept treatment caused by neuroborreliosis. Br J Dermatol 163: 228-30
  • Molin S, Ruzicka T, Prinz JC (2010) Borreliosis mimicking lupus-like syndrome during infliximab treatment. Clin Exp Dermatol 35: 631-3
  • Molin S, Vollmer S, Weiss EH, Ruzicka T, Prinz JC (2009) Filaggrin mutations may confer susceptibility to chronic hand eczema characterized by combined allergic and irritant contact dermatitis. Br J Dermatol 161: 801-7
  • Molin S, Vollmer S, Weiss EH, Weisenseel P, Ruzicka T, Prinz JC (2011) Deletion of the late cornified envelope genes LCE3B and LCE3C may promote chronic hand eczema with allergic contact dermatitis. J Investig Allergol Clin Immunol 21: 472-9
  • Prinz J, Braun-Falco O, Meurer M, Daddona P, Reiter C, Rieber P, Riethmuller G (1991) Chimaeric CD4 monoclonal antibody in treatment of generalised pustular psoriasis. Lancet 338: 320-1
  • Prinz JC (1997) Psoriasis vulgaris, streptococci and the immune system: a riddle to be solved soon? Scand J Immunol 45: 583-6
  • Prinz JC (1999) Which T cells cause psoriasis? Clin Exp Dermatol 24: 291-5
  • Prinz JC (2001) Psoriasis vulgaris--a sterile antibacterial skin reaction mediated by cross-reactive T cells? An immunological view of the pathophysiology of psoriasis. Clin Exp Dermatol 26: 326-32
  • Prinz JC (2003) [Latest aspects in psoriasis pathogenesis]. Hautarzt 54: 209-14
  • Prinz JC (2003) The role of T cells in psoriasis. J Eur Acad Dermatol Venereol 17: 257-70
  • Prinz JC (2004) Disease mimicry--a pathogenetic concept for T cell-mediated autoimmune disorders triggered by molecular mimicry? Autoimmun Rev 3: 10-5
  • Prinz JC (2004) [New aspects in the pathogenesis of psoriasis]. J Dtsch Dermatol Ges 2: 448-56; quiz 457
  • Prinz JC (2010) [Biologics. New drugs, new adverse reactions]. Hautarzt 61: 668-75
  • Prinz JC (2011) Autoimmune-like syndromes during TNF blockade: does infection have a role? Nat Rev Rheumatol 7: 429-34
  • Prinz JC, Gross B, Vollmer S, Trommler P, Strobel I, Meurer M, Plewig G (1994) T cell clones from psoriasis skin lesions can promote keratinocyte proliferation in vitro via secreted products. Eur J Immunol 24: 593-8
  • Prinz JC, Kutasi Z, Weisenseel P, Poto L, Battyani Z, Ruzicka T (2009) "Borrelia-associated early-onset morphea": a particular type of scleroderma in childhood and adolescence with high titer antinuclear antibodies? Results of a cohort analysis and presentation of three cases. J Am Acad Dermatol 60: 248-55
  • Prinz JC, Vollmer S, Boehncke WH, Menssen A, Laisney I, Trommler P (1999) Selection of conserved TCR VDJ rearrangements in chronic psoriatic plaques indicates a common antigen in psoriasis vulgaris. Eur J Immunol 29: 3360-8
  • Vollmer S, Menssen A, Prinz JC (2001) Dominant lesional T cell receptor rearrangements persist in relapsing psoriasis but are absent from nonlesional skin: evidence for a stable antigen-specific pathogenic T cell response in psoriasis vulgaris. J Invest Dermatol 117: 1296-301
  • Vollmer S, Menssen A, Trommler P, Schendel D, Prinz JC (1994) T lymphocytes derived from skin lesions of patients with psoriasis vulgaris express a novel cytokine pattern that is distinct from that of T helper type 1 and T helper type 2 cells. Eur J Immunol 24: 2377-82
  • Weisenseel P, Kuznetsov AV, Prinz JC (2007) [Implementation of the S3-guidelines for systemic and UV therapy of moderate to severe psoriasis vulgaris by an algorithm]. J Dtsch Dermatol Ges 5: 683-8
  • Weisenseel P, Laumbacher B, Besgen P, Ludolph-Hauser D, Herzinger T, Roecken M, Wank R, Prinz JC (2002) Streptococcal infection distinguishes different types of psoriasis. J Med Genet 39: 767-8
  • Weisenseel P, Martin S, Partscht K, Messer G, Prinz JC (2007) Relevance of the low-affinity type of the Fcgamma-receptor IIIa-polymorphism in bullous pemphigoid. Arch Dermatol Res 299: 163-4
  • Weisenseel P, Prinz JC (2005) Incidental detection of S. pyogenes-DNA in psoriatic skin by PCR. Arch Dermatol Res 296: 573-6
  • Weisenseel P, Prinz JC (2006) Sequential use of infliximab and etanercept in generalized pustular psoriasis. Cutis 78: 197-9

 

 
 

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